A CONSIDERATION TO IMMUNE DOSES OF STAPHYLOCOCCAL ENTEROTOXIN B TO RABBITS
نویسندگان
چکیده
منابع مشابه
A DNA Spiegelmer to staphylococcal enterotoxin B.
Bacterial staphylococcal enterotoxin B is involved in several severe disease patterns and it was therefore used as a target for the generation of biologically stable mirror-image oligonucleotide ligands, so called Spiegelmers. The toxin is a 28 kDa protein consisting of 239 amino acids. Since the full-length protein is not accessible to chemical peptide synthesis, a stable domain of 25 amino ac...
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Staphylococcal enterotoxins are 23- to 29-kDa polypeptides in the bacterial superantigen protein family. Clinical symptoms from intoxication with staphylococcal enterotoxins vary by exposure route. Ingestion results in gastrointestinal symptoms, and inhalation results in fever as well as pulmonary and gastrointestinal symptoms. Review of occupational exposures at the U.S. Army Medical Research ...
متن کاملHomogeneous enzyme immune assay for staphylococcal enterotoxin B.
A simple homogeneous enzyme immune assay was developed for detection of staphylococcal enterotoxin B by a conjugate of beta-amylase coupled with the enterotoxin.
متن کاملSulfasalazine Attenuates Staphylococcal Enterotoxin B-Induced Immune Responses
Staphylococcal enterotoxin B (SEB) and related exotoxins are important virulence factors produced by Staphylococcus aureus as they cause human diseases such as food poisoning and toxic shock. These toxins bind directly to cells of the immune system resulting in hyperactivation of both T lymphocytes and monocytes/macrophages. The excessive release of proinflammatory cytokines from these cells me...
متن کاملRole of endogenous interleukin-12 in immune response to staphylococcal enterotoxin B in mice.
In the present study, the roles of interleukin 12 (IL-12) and IL-18 and their possible interaction during superantigen-induced responses were studied by injection of staphylococcal enterotoxin B (SEB) into mice. These data suggest that the role of IL-12 in SEB-induced responses is limited to sustaining gamma interferon release by an IL-18-independent mechanism.
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ژورنال
عنوان ژورنال: Japanese Journal of Medical Science and Biology
سال: 1974
ISSN: 0021-5112,1884-2828
DOI: 10.7883/yoken1952.27.309